Upper Airway Obstruction
Rhoda Wynn
Ari J. Goldsmith
Upper airway obstruction must be differentiated from respiratory distress caused by pulmonary, neurologic, cardiovascular, and esophageal disorders. Anatomically, the upper airway consists of the air-conducting passages from the mouth and nose to the carina. The causes of respiratory obstruction in children and adults are discussed separately because of the relative prevalence of causes; upper airway obstruction in adults tends to be inflammatory or neoplastic, whereas in children it tends to be inflammatory or congenital.
The anatomy of the upper airway is different in children than adults. The caliber of the air-conducting passages is larger in adults, allowing tolerance of larger obstructions. While the narrowest part of the adult airway is the glottis, in children it is the subglottis; even minimal edema or inflammation of the subglottis in a child can cause marked obstruction and severe distress. Newborns are obligate nasal breathers because they have a high-riding larynx. Any nasal obstruction can produce cyclical cyanosis. The baby cries once the cyanosis becomes uncomfortable and is able to breath through the mouth.
Signs and symptoms of airway obstruction depend on the site of obstruction rather than the precise cause. Because the site of obstruction plays an important role in treatment, this chapter offers a diagnostic approach based on the anatomic site of obstruction (Tables 29-1 and 29-2).
MEDICAL HISTORY AND PHYSICAL EXAMINATION
A careful medical history is essential in the evaluation of upper airway obstruction and helps to categorize the nature of the obstruction. The predominant symptoms of airway dysfunction elicited during a careful history interview are stridor, sturdor, dyspnea, cough, aspiration, hoarseness, and dysphagia and odynophagia. Depending on the degree of upper airway obstruction and ability of the patient to compensate, the signs and symptoms may vary from severe stridor with air hunger to mild sturdor.
The character of noisy breathing in relation to the respiratory cycle helps localize the site of obstruction. When the obstruction is supraglottic or oropharyngeal, muffled, rattling, noisy breathing (sturdor) occurs and may be partly relieved when the tongue is pulled forward. Supraglottic obstruction produces inspiratory stridor. Glottic obstruction also tends to produce inspiratory stridor, but usually with a hoarse or breathy voice. However, a normal voice may still be present if the vocal cords remain in a median position due to bilateral vocal cord paralysis. Subglottic obstruction produces inspiratory and expiratory stridor. Intrathoracic obstruction produces expiratory wheezing.
The onset, frequency, duration of noisy breathing and the presence of cyanosis should be determined. Associations with feeding, agitation, exercise, sleep, and position changes should be
sought. For instance, a child with feeding difficulties, prolonged feeding times, or regurgitation with stridor that worsens in the supine position is likely to have gastroesophageal reflux disease. The degree of dyspnea and anxiety helps determine the acuity of the obstruction. A cough is pathologic when it increases in frequency or is associated with production of secretions. Hoarseness is more ominous when it is progressive rather than intermittent.
sought. For instance, a child with feeding difficulties, prolonged feeding times, or regurgitation with stridor that worsens in the supine position is likely to have gastroesophageal reflux disease. The degree of dyspnea and anxiety helps determine the acuity of the obstruction. A cough is pathologic when it increases in frequency or is associated with production of secretions. Hoarseness is more ominous when it is progressive rather than intermittent.
TABLE 29-1. Causes of adult upper airway obstruction according to site | ||||||||||||||||||||||||||||||||||
---|---|---|---|---|---|---|---|---|---|---|---|---|---|---|---|---|---|---|---|---|---|---|---|---|---|---|---|---|---|---|---|---|---|---|
|