Orbital Trauma
Parag D. Gandhi, MD, FACS
ORBITAL FLOOR FRACTURES
Pathophysiology
Facial trauma resulting in direct blunt injury to the globe and adnexa, creating retropulsive positive pressure in orbit versus force propagating from the inferior orbital rim along the floor
Downward buckling of the thinnest portion of the maxilla most often, comprising floor of orbit and roof of underlying maxillary sinus
Inferior rectus muscle, inferior oblique muscle, perimuscular fascia, or orbital fat can become impinged or entrapped along the fracture site.
Symptoms can include pain with eye movement and/or double vision.
Associated findings may include enophthalmos or hypoglobus, and numbness in V2 distribution over the cheek.
Vision loss may result from trauma.
Management Options
Up to 30% of orbital fractures have concomitant globe injury; therefore, a complete ophthalmologic examination is necessary.
Ocular injuries must be managed first.
Orbital computed tomography (CT) for confirmation of floor fracture defect
Observation
Many fractures heal without surgery and have no ophthalmic or adnexal sequelae.
Recent literature reflects a shift to later repair of isolated orbital floor fractures.
Surgical repair is often indicated to prevent late sequelae of enophthalmos and/or double vision. Preoperative antibiotics, avoiding nose blowing, and oral corticosteroids are often employed.
Indications for Surgery
Double vision, usually in upgaze or downgaze, but could be in any direction depending on what tissues are entrapped.
Caution initially as double vision could be due to traumatic edema, which will resolve within 1 to 2 weeks, suggesting the need to wait on finalizing the decision
Orbital CT with axial, coronal, and sagittal views with thin cuts demonstrating fracture defect greater than 50% of floor (Figure 33.1)
Enophthalmos greater than 2 mm due to fracture
Primary indication for immediate surgical repair is significant muscle entrapment, which could be in a narrower fracture defect, causing marked double vision and possible vasovagal effects.
Surgical Description
Preoperative selection of floor repair implant. Many alloplastic options including resorbable sheets, thin plastic sheeting, titanium mesh, porous polyethylene sheets, combination titanium and porous polyethylene sheets, preformed titanium or combination implants, and autografted calvarial bone
Selection based on size of fracture, area of stable bone, degree of support needed, and surgeon comfort with various materials
Preoperative oxymetazoline nasal spray to decongest sinus passages on both sides
General anesthesia with endotracheal intubation generally safer due to risk of blood in the airway
Preoperative antibiotics are often used.
Intraoperative intravenous (IV) corticosteroids can reduce postoperative edema.
Inject local infiltrative anesthesia into the surgical area.
Make a transconjunctival incision 5 mm below the inferior border of the tarsus and dissect to the arcus marginalis remaining preseptal, if possible, to keep the fat posterior.
Using a Freer elevator, open the periosteum widely along the inferior orbital rim lateral to medial for exposure, and dissect posteriorly along the floor to identify the anterior, medial, and lateral extent of fracture defect.
Gently reduce the soft tissue back into the orbit, taking care to avoid infraorbital neurovascular structures. Retract the orbital contents under a malleable retractor to
identify the posterior edge of the fracture and isolate the posterior ledge. Consider herniated bone fragment removal from underlying maxillary sinus, especially if the bone flap is obstructing sinus outflow.
Contour and place an implant to cover the fracture site on all edges, especially the posterior ledge.
Confirm hemostasis with bipolar cautery and/or injectable hemostatic matrix. Avoid bipolar over or near nerves.
Perform forced duction testing of globe to confirm full movement and no residual impingement.
Consider anterior fixation of implant along the inferior rim with one to two titanium mini screws if using a titanium mesh based implant or over a large fracture defect.
Close the periosteum over the inferior rim with 4-0 polyglactin sutures.
Remove all instruments and any packing.
Monitor the pupil for dilation during the repair and alleviate pressure on the globe when needed.
Postoperative Management
Steroid antibiotic ointment to incisions three times per day for 1 week and then daily for 1 week
Cool compresses for 48 hours
Elevate head of bed.
Avoid driving for 1 week.
Avoid bending, lifting, and strenuous activity for 2 weeks.
Avoid nose blowing for 3 weeks.
Follow up at 1 week, 3 weeks, 2 months, and sooner as needed.
Prognosis
Resolution of double vision and enophthalmos is good for early to medium term repairs with minimal scarring.
Prognosis for resolution guarded if late repair with scarring and/or impingement of inferior rectus muscle or perimuscular fascia/fat
Postoperative V2 numbness usually resolves with time, but can be long term or permanent.
Small risk of vision loss or blindness from surgery should be discussed during preoperative informed consent.
ORBITAL MEDIAL WALL FRACTURES
Pathophysiology
Facial trauma resulting in direct blunt injury to the globe and adnexa, creating retropulsive positive pressure in orbit to ethmoids/lamina papyracea (thinnest bones of orbit)
Outward buckling of the medial wall into adjacent anterior and/or posterior ethmoid air cells
Medial rectus muscle, perimuscular fascia, or orbital fat can become impinged or entrapped along the fracture site.
Symptoms can include pain with eye movement and/or double vision.
Associated findings may include enophthalmos of the affected globe.
Vision loss may also result from trauma.
Management Options
Must rule out concomitant globe injury; therefore, a complete ophthalmologic examination is necessary.
Ocular injuries must be managed first.
Orbital CT for confirmation of medial wall fracture defect
Observation
Many fractures heal without surgery and have no ophthalmic or adnexal sequelae.
Surgical repair is sometimes indicated to prevent late sequelae of enophthalmos and/or double vision. Preoperative antibiotics, avoiding nose blowing, and oral corticosteroids are often employed.
Indications for Surgery
Orbital CT with axial and coronal thin cuts showing moderate to large fracture greater than 50% of medial wall area and herniation of orbital soft tissue into defect (Figure 33.2).
Marked enophthalmos due to fracture
Horizontal double vision
Surgical Description
Preoperative selection of alloplastic medial wall implant. Options include resorbable sheets, thin plastic sheeting, titanium mesh, porous polyethylene sheets, and combination titanium and porous polyethylene sheets. Selection depends on size of defect and need for anterior fixation.
Preoperative oxymetazoline nasal spray to decongest sinus passages on both sides
General anesthesia with endotracheal intubation is generally safer due to risk of blood in the airway.
Preoperative antibiotics are often used.
Intraoperative IV corticosteroids can reduce postoperative edema.
Inject local infiltrative anesthesia into the surgical area.
Consider intranasal packing of cottonoid sponges soaked in oxymetazoline.
FIGURE 33.2. Orbital and facial bone CT axial view showing large left medial wall fracture, greater than 50% of wall, with herniated orbital soft tissue into anterior and posterior ethmoid sinus.
Use a transconjunctival postcaruncular approach to the medial orbital rim remaining posterior to the lacrimal structures and immediately subconjunctival, if possible, to keep fat out of the operative field.
Using a Freer elevator, open the periosteum widely along the medial orbital rim for exposure, and dissect posteriorly along the medial wall to identify the anterior, superior, and inferior extent of the fracture defect.
Gently reduce the soft tissue back into the orbit, taking care to avoid the ethmoidal vascular structures.
Identify and isolate the posterior ledge of the fracture. Consider herniated bone fragment removal from ethmoids if interfering with insertion and correct placement of implant.
Contour and place an implant to cover the fracture site on all edges, especially the posterior ledge.
Confirm hemostasis with bipolar cautery and/or injectable hemostatic matrix. Avoid bipolar over or near nerves.
Perform forced duction testing of the globe to confirm full movement and no residual impingement.
Close the medial conjunctival incision with 6-0 plain gut sutures.
Remove all instruments and any packing.
Great care must be taken to avoid the anterior and posterior ethmoidal arteries along the frontoethmoidal suture line approximately 5 mm superior to the medial canthus and 24 mm and 36 mm posterior to the anterior lacrimal crest, respectively.
Postoperative Management
Steroid antibiotic ointment to incisions three times per day for 1 week and then daily for 1 week
Cool compresses for 48 hours
Elevate head of bed.
Avoid driving for 1 week.
Avoid bending, lifting, and strenuous activity for 2 weeks.
Avoid nose blowing for 3 weeks.
Follow up at 1 week, 3 weeks, 2 months, and sooner as needed.
Prognosis
Resolution of double vision and enophthalmos good for early to medium term repairs with minimal scarring.
Prognosis for resolution guarded if late repair with scarring and/or impingement of medial rectus muscle or perimuscular fascia/fat
A small risk of damage to anterior lacrimal structures should be discussed preoperatively with patients.
A small risk of vision loss or blindness from surgery should be discussed during preoperative informed consent.
ZYGOMATICOMAXILLARY COMPLEX (ZMC) FRACTURES
Pathophysiology
Usually more severe facial trauma such as from a motor vehicle accident (MVA) or a fall from a height resulting in severe blunt injury to facial skeleton and adnexa, creating retropulsion of zygoma into the facial skeleton, or separation often along bony suture lines at three or more locations (hence, the name “tripod” or “trimalar” fracture is actually a misnomer).
Downward displacement of lateral canthus along with downward or upward buckling of the thinnest portion of the maxilla comprising floor of orbit and roof of underlying maxillary sinus.
Inferior rectus muscle, inferior oblique muscle, perimuscular fascia, or orbital fat can become impinged or entrapped along the fracture site.
Symptoms can include pain with eye movement and/or double vision.
Associated findings may include facial deformity, step deformity along orbital rim, displacement of affected globe, and numbness in V2 distribution over the cheek.
Vision loss may result from trauma.
Management Options
Up to 30% of orbital fractures have concomitant globe injury; therefore, a complete ophthalmologic examination is necessary.
Ocular injuries must be managed first.
Orbital and facial bone CT for confirmation of displaced zygoma and concomitant orbital floor defect
Observation
If fracture present but stable and nondisplaced, may heal without ophthalmic or adnexal sequelae
Surgical repair is usually indicated to prevent late sequelae of facial and adnexal deformity, globe displacement, and/or double vision. Preoperative antibiotics, avoiding nose blowing, and oral corticosteroids are often employed.
Sequencing of treatment options with stabilization of concomitant ophthalmic injuries may be required first.
Indications for Surgery
Facial deformity
Lateral canthal dystopia
Double vision, usually in upgaze or downgaze, but could be in any direction depending on which tissues are entrapped
Caution initially as double vision could be due to traumatic edema, which will resolve within 1 to 2 weeks. Zygomaticomaxillary complex fractures should be repaired within 2 weeks to prevent early osseous malunion.
Orbital and facial CT with axial, coronal, and sagittal views with thin cuts demonstrating zygoma displacement at three or more locations (Figure 33.3)
Fracture defect of the orbital floor may appear artificially smaller in telescoping injuries and may require intraoperative decision for repair.
Surgical Description
Preoperative selection of titanium implant plating set. Many options including size, curve, and thickness of plates (usually small to medium sized)
Preoperative oxymetazoline nasal spray to decongest sinus passages on both sides. General anesthesia with endotracheal intubation generally safer due to risk of blood in the airway.
Preoperative antibiotics are often used.
Intraoperative IV corticosteroids can reduce postoperative edema.
Inject local infiltrative anesthesia to surgical areas, anticipating multiple sites of exposure. Consider intranasal packing of cottonoid sponges soaked in oxymetazoline.
Make a transconjunctival incision 5 mm below inferior border of tarsus and dissect to the arcus marginalis remaining preseptal, if possible, to keep the fat posterior.
Perform a lateral canthotomy with inferior cantholysis to expose the thickest portion of the malar eminence. Using a Freer elevator, open the periosteum widely along the inferior orbital rim lateral to medial for exposure and dissect posteriorly along the
floor to identify the anterior, medial, and lateral extent of the fracture defect. Next, from the arcus marginalis, dissect anteriorly and laterally to expose the fracture site and inferior orbital rim portions of the maxilla and zygoma for reduction and plating.
Place a threaded reduction screw, such as a Carroll-Girard screw, in the thickest portion of the zygoma for bony reduction (Figure 33.4).
Next, make an incision in the lateral upper eyelid crease or in an existing traumatic laceration for access to the zygomaticofrontal suture line. Dissect down to the lateral orbital rim and use a Freer elevator to open the periosteum from the external lateral orbital rim and the lateral orbital wall to identify the zygomaticosphenoid suture line.
Use a reduction tool to fully reposition the zygoma, confirming proper alignment along the inferior orbital rim, lateral orbital rim, and exposed lateral orbital wall.
Before plating the zygoma, perform a subperiosteal dissection posteriorly along the orbital floor to identify the anterior, medial, and lateral extent of the fracture defect, if present.
Gently reduce any soft tissue back into the orbit as needed, taking care to avoid infraorbital neurovascular structures.
Confirm all bony and soft tissue reduction.
Plate the zygoma to the surrounding stable bone at two or more points of fixation. Secure at least two screws through the plate holes on either side of the fracture and one to two plate holes over the fracture site as needed (Figure 33.5).
For the orbital floor portion, identify the posterior edge of the fracture and isolate the posterior ledge. Contour and place an implant to cover all edges of the fracture site, especially the posterior ledge.
Confirm hemostasis with bipolar cautery and/or injectable hemostatic matrix. Avoid bipolar over or near nerves.
Perform forced duction testing of the globe to confirm full movement with no residual impingement.
Close the periosteum over the inferior and lateral orbital rims with 4-0 polyglactin sutures.
Perform midfacial soft tissue resuspension for proper postoperative midfacial support as discussed elsewhere in this text.
Perform lateral canthal resuspension and skin closure as discussed elsewhere in this text.
Remove all instruments and any packing.
Occasionally, an intraoral approach through a Caldwell-Luc incision with the added visualization from below is needed to align the teeth and lower portion of the zygomaticomaxillary suture line.
Postoperative Management
Steroid antibiotic ointment to incisions three times per day for 1 week and then daily for 1 week
Cool compresses for 48 hours
Elevate head of bed.
Avoid driving for 1 week.
Avoid bending, lifting, and strenuous activity for 2 weeks.
Avoid nose blowing for 3 weeks.
Follow up at 1 week, 3 weeks, 2 months, and sooner as needed.
Prognosis
Resolution of facial and adnexal deformity, any double vision, or globe displacement good for early repairs within 2 weeks with minimal scarring
Prognosis for proper reduction and resolution of all defects and deformities guarded if delayed repair with scarring and/or impingement of rectus muscles or perimuscular fascia/fatStay updated, free articles. Join our Telegram channel
Full access? Get Clinical Tree